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Synaptic activity-dependent changes in the hippocampal palmitoylome

Synaptic activity-dependent changes in the hippocampal palmitoylome

Synaptic activity-dependent changes in the hippocampal palmitoylome. Nasseri GG, Matin N, Wild AR, Tosefsky K, Flibotte S, Stacey RG, Hollman RB, Foster LJ, Bamji SX. (2022) Science Signaling. Dec 6; 15(763). PMID: 36473050

Exploring the expression patterns of palmitoylating and de-palmitoylating enzymes in the mouse brain using the curated RNA-seq database BrainPalmSeq

Exploring the expression patterns of palmitoylating and de-palmitoylating enzymes in the mouse brain using the curated RNA-seq database BrainPalmSeq

Exploring the expression patterns of palmitoylating and de-palmitoylating enzymes in the mouse brain using the curated RNA-seq database BrainPalmSeq. Wild AR, Hogg PW, Flibotte S, Nasseri GG, Hollman RB, Abazari D, Haas K, Bamji SX. (2022) Elife. Jul 12;11:e75804. PMID: 35819139

The X-Linked Intellectual Disability Gene Zdhhc9 Is Essential for Dendrite Outgrowth and Inhibitory Synapse Formation.

The X-Linked Intellectual Disability Gene Zdhhc9 Is Essential for Dendrite Outgrowth and Inhibitory Synapse Formation.

The X-Linked Intellectual Disability Gene Zdhhc9 Is Essential for Dendrite Outgrowth and Inhibitory Synapse Formation. Shimell JJ, Shah BS, Cain SM, Thouta S, Kuhlmann N, Tatarnikov I, Jovellar DB, Brigidi GS, Kass J, Milnerwood AJ, Snutch TP, Bamji SX. (2019) Cell Reports. Nov 19;29(8):2422-2437. PMID: 31747610

Cadherins mediate cocaine-induced synaptic plasticity and behavioral conditioning.

Cadherins mediate cocaine-induced synaptic plasticity and behavioral conditioning.

Cadherins mediate cocaine-induced synaptic plasticity and behavioral conditioning. Mills F*, Globa AK*, Liu S, Cowan CM, Mobasser M, Philips AG, Borgland SL, Bamji SX. (2017) Nature Neuroscience. Apr;20(4):540-549.PMID: 28192395

Consumption of palatable food primes food approach behavior by rapidly increasing synaptic density in the VTA.

Consumption of palatable food primes food approach behavior by rapidly increasing synaptic density in the VTA.

Consumption of palatable food primes food approach behavior by rapidly increasing synaptic density in the VTA. Liu S, Globa AK, Mills F, Naef L, Qiao M, Bamji SX, Borgland SL. (2016) Proceedings of the National Academy of Sciences U.S.A. March 1:113(9):2520-2525.PMID: 26884159

Use-dependent activation of neuronal Kv1.2 channel complexes.

Use-dependent activation of neuronal Kv1.2 channel complexes.

Use-dependent activation of neuronal Kv1.2 channel complexes. Baronas VA, McGuinness BR Vilin YY, Brigidi SG, Bamji SX, Yang R, Kurata T. (2015) Journal of Neuroscience. Feb 25;35(8):4518-13.PMID: 25716850

Activity-Regulated Trafficking of the Palmitoyl-Acyl Transferase DHHC5.

Activity-Regulated Trafficking of the Palmitoyl-Acyl Transferase DHHC5.

Activity-Regulated Trafficking of the Palmitoyl-Acyl Transferase DHHC5. Brigidi S, Santyr B, Shimell J, Jovellar B, Bamji SX. (2015) Nature Communications. Sep 3;6:8200.PMID: 26334723​

Cognitive flexibility and long-term depression (LTD) are impaired following β-catenin stabilization in vivo.

Cognitive flexibility and long-term depression (LTD) are impaired following β-catenin stabilization in vivo.

Cognitive flexibility and long-term depression (LTD) are impaired following β-catenin stabilization in vivo. Mills F, Bartlett TE, Dissing-Olesen L, Wisniewska MB, Kuznicki J, Macvicar BA, Wang YT, Bamji SX. (2014) Proceedings of the National Academy of Sciences. U.S.A. Jun 10;111(23):8631-6.PMID: 24912177

Palmitoylation of δ-catenin by DHHC5 mediates activity-induced synapse plasticity.

Palmitoylation of δ-catenin by DHHC5 mediates activity-induced synapse plasticity.

Palmitoylation of δ-catenin by DHHC5 mediates activity-induced synapse plasticity. Brigidi GS, Sun Y, Beccano-Kelly D, Pitman K, Mobasser M, Borgland SL, Milnerwood AJ, Bamji SX. (2014) Nature Neuroscience. Apr;17(4):522-32.PMID: 24562000

Hyperinsulinemia drives diet-induced obesity independently of brain insulin production.

Hyperinsulinemia drives diet-induced obesity independently of brain insulin production.

Hyperinsulinemia drives diet-induced obesity independently of brain insulin production. Mehran AE, Templeman NM, Brigidi GS, Lim GE, Chu KY, Hu X, Botezelli JD, Asadi A, Hoffman BG, Kieffer TJ, Bamji SX, Clee SM, Johnson JD. (2013) Cell Metabolism. Dec 5;16(6):723-37.PMID: 23217255Y.